logging in or signing up A CASE OF SUDDEN UNILATERAL LOSS OF VISION tnsood Download Post to : URL : Related Presentations : Share Add to Flag Embed Email Send to Blogs and Networks Add to Channel Uploaded from authorPOINT lite Insert YouTube videos in PowerPont slides with aS Desktop Copy embed code: (To copy code, click on the text box) Embed: URL: Thumbnail: WordPress Embed Customize Embed The presentation is successfully added In Your Favorites. Views: 255 Category: Entertainment License: All Rights Reserved Like it (0) Dislike it (0) Added: September 17, 2010 This Presentation is Public Favorites: 0 Presentation Description No description available. Comments Posting comment... Premium member Presentation Transcript CASE PRESENTATION A Case of Sudden U/L loss of vision : CASE PRESENTATION A Case of Sudden U/L loss of vision Dr Tarun sood Patient Particulars : Patient Particulars Anil Kumar Das 27 yr old Sep of 220 Med Regt Reported to Eye OPD on 27 Mar 08 Chief Complaints: Pain -06 days Sudden loss of vision RE -05 days History : History No h/o trauma No h/o fever No h/o exposure to snow No h/o Drug intake of long standing duration No h/o TB/DM/HTN H/O TAKING ALCOHOL(allegedly issued from 403 Fd Amb)on22Mar–Holi(Saturday) continued… : continued… Pt had mild Pain Rt Eye that day He woke up the next day with inability to see anything with his Rt eye(Sunday) He reported to 403 Fd Amb the next day (Monday) whence he was transferred to 153 GH for Ophthal opinion Reported here on 27 Mar 08 Ocular Examination : Ocular Examination DVA RE-HMCF LE-6/6 Both Eyes White and quiet RE – Pain and tenderness present on ocular movements( more so on looking up and medially) LE – No Pain/Tenderness Continued… : Continued… RE Ant Seg:Cornea clear, AC-Normal,RAPD +nt Post Seg:Posterior Vitreous Haze +nt Disc hyperemic blurred margins peripapillary edema No Haemorrhages No Exudates Macula- Extensive Macular edema with evidence of macular fan Continued… : Continued… RE COLOUR VISION Defective AMSLER CHARTING Normal LIGHT BRIGHTNESS Diminished VISUAL FIELDS(CFT) Central scotoma LE All investigations WNL DIFFERENTIAL DIAGNOSIS : DIFFERENTIAL DIAGNOSIS Papilloedema Papillitis Congenital Disc Anomalies Medullated Nerve Fibres Orbital lesions Optic Disc Drusen Malignant cell infiltration of Nerve head Ocular Hypotony Differentiating FeaturesSYMPTOMS : Differentiating FeaturesSYMPTOMS PAPILLOEDEMA Usually B/L Transient attacks of blurred vision No pain and tenderness PAPILLITIS Usually U/L Marked loss of vision of sudden onset Pain and tenderness with ocular movements Differentiating FeaturesSIGNS : Differentiating FeaturesSIGNS Pupillary Rx,Colour visionand contrast sensitivity-Normal Media clear, Disc hyperemic, margins blurred,2to6D swelling of disc Peripapillary oedema, engorged veins,RetHges exudates,Macular star+nt Fields-Enlarged BlindSpot MarcusGunn Pupil, Colour vision defective, Contrast sensitivity decreased Media-posterior vitreous haze,Disc hyperemic,blurred margins,mild swelling Peripapillary edema,No Hges,exudates,Macular edema +nt Central /Centrocaecal scotoma Optic Neuritis : Inflammation of optic nerve : Optic Neuritis : Inflammation of optic nerve Papillitis Neuroretinitis Retrobulbar Neuritis Aetiology of Optic Neuritis : Aetiology of Optic Neuritis Idiopathic Demyelinating disorders Asso with infections:Local,Systemic Immune mediated disorders Metabolic disorders Toxic Optic Neuritis Clinical Features : Clinical Features Loss of vision Periocular discomfort made worse on moving the eye Frontal headache Tenderness of globe Diminished Visual Acuity Marcus Gunn Pupil Colour vision defects Visual field defects Diminished light brightness sensitivity Points in favour of papillitis : Points in favour of papillitis U/L Pain on eye movement Severely diminished vision RAPD Defective Colour Vision Diminished light brightness Ophthalmoscopic Picture : Ophthalmoscopic Picture Posterior Vitreous Haze Disc-Hyperemic, Oedematous with blurred margins No Hges/Exudates Macular edema Management : Management Investigations Hb, TLC, DLC, ESR BS-F,PP ,Lipid profile Serum Ca,K,Urea,Creatinine Urine exam ECG Chest X-ray PULSE STEROID THERAPY I/V Methyl Prednisolone 250 mg 6 hrly for 3 days foll by Oral Prednisolone 1mg/kg/day for 11 days Taper off over 3 days Follow up : Follow up 31 Mar 08 – Vision improved drastically to 4/60 LE 01 April 08 – Vision improved further to 6/36 03 April 08 – Vision improved to 6/18 05 April 08 – Vision improved to 6/9p TODAY O7 APRIL VISION IS 6/6P Conclusion : Conclusion Optic Neuritis is a serious condition which if not treated immediately can lead to permanent visual loss. ANY PATIENT COMPLAINING OF SUDDEN DETERIORATION OF VISION should be critically examined , thoroughly investigated and promptly managed. You do not have the permission to view this presentation. In order to view it, please contact the author of the presentation.
A CASE OF SUDDEN UNILATERAL LOSS OF VISION tnsood Download Post to : URL : Related Presentations : Share Add to Flag Embed Email Send to Blogs and Networks Add to Channel Uploaded from authorPOINT lite Insert YouTube videos in PowerPont slides with aS Desktop Copy embed code: (To copy code, click on the text box) Embed: URL: Thumbnail: WordPress Embed Customize Embed The presentation is successfully added In Your Favorites. Views: 255 Category: Entertainment License: All Rights Reserved Like it (0) Dislike it (0) Added: September 17, 2010 This Presentation is Public Favorites: 0 Presentation Description No description available. Comments Posting comment... Premium member Presentation Transcript CASE PRESENTATION A Case of Sudden U/L loss of vision : CASE PRESENTATION A Case of Sudden U/L loss of vision Dr Tarun sood Patient Particulars : Patient Particulars Anil Kumar Das 27 yr old Sep of 220 Med Regt Reported to Eye OPD on 27 Mar 08 Chief Complaints: Pain -06 days Sudden loss of vision RE -05 days History : History No h/o trauma No h/o fever No h/o exposure to snow No h/o Drug intake of long standing duration No h/o TB/DM/HTN H/O TAKING ALCOHOL(allegedly issued from 403 Fd Amb)on22Mar–Holi(Saturday) continued… : continued… Pt had mild Pain Rt Eye that day He woke up the next day with inability to see anything with his Rt eye(Sunday) He reported to 403 Fd Amb the next day (Monday) whence he was transferred to 153 GH for Ophthal opinion Reported here on 27 Mar 08 Ocular Examination : Ocular Examination DVA RE-HMCF LE-6/6 Both Eyes White and quiet RE – Pain and tenderness present on ocular movements( more so on looking up and medially) LE – No Pain/Tenderness Continued… : Continued… RE Ant Seg:Cornea clear, AC-Normal,RAPD +nt Post Seg:Posterior Vitreous Haze +nt Disc hyperemic blurred margins peripapillary edema No Haemorrhages No Exudates Macula- Extensive Macular edema with evidence of macular fan Continued… : Continued… RE COLOUR VISION Defective AMSLER CHARTING Normal LIGHT BRIGHTNESS Diminished VISUAL FIELDS(CFT) Central scotoma LE All investigations WNL DIFFERENTIAL DIAGNOSIS : DIFFERENTIAL DIAGNOSIS Papilloedema Papillitis Congenital Disc Anomalies Medullated Nerve Fibres Orbital lesions Optic Disc Drusen Malignant cell infiltration of Nerve head Ocular Hypotony Differentiating FeaturesSYMPTOMS : Differentiating FeaturesSYMPTOMS PAPILLOEDEMA Usually B/L Transient attacks of blurred vision No pain and tenderness PAPILLITIS Usually U/L Marked loss of vision of sudden onset Pain and tenderness with ocular movements Differentiating FeaturesSIGNS : Differentiating FeaturesSIGNS Pupillary Rx,Colour visionand contrast sensitivity-Normal Media clear, Disc hyperemic, margins blurred,2to6D swelling of disc Peripapillary oedema, engorged veins,RetHges exudates,Macular star+nt Fields-Enlarged BlindSpot MarcusGunn Pupil, Colour vision defective, Contrast sensitivity decreased Media-posterior vitreous haze,Disc hyperemic,blurred margins,mild swelling Peripapillary edema,No Hges,exudates,Macular edema +nt Central /Centrocaecal scotoma Optic Neuritis : Inflammation of optic nerve : Optic Neuritis : Inflammation of optic nerve Papillitis Neuroretinitis Retrobulbar Neuritis Aetiology of Optic Neuritis : Aetiology of Optic Neuritis Idiopathic Demyelinating disorders Asso with infections:Local,Systemic Immune mediated disorders Metabolic disorders Toxic Optic Neuritis Clinical Features : Clinical Features Loss of vision Periocular discomfort made worse on moving the eye Frontal headache Tenderness of globe Diminished Visual Acuity Marcus Gunn Pupil Colour vision defects Visual field defects Diminished light brightness sensitivity Points in favour of papillitis : Points in favour of papillitis U/L Pain on eye movement Severely diminished vision RAPD Defective Colour Vision Diminished light brightness Ophthalmoscopic Picture : Ophthalmoscopic Picture Posterior Vitreous Haze Disc-Hyperemic, Oedematous with blurred margins No Hges/Exudates Macular edema Management : Management Investigations Hb, TLC, DLC, ESR BS-F,PP ,Lipid profile Serum Ca,K,Urea,Creatinine Urine exam ECG Chest X-ray PULSE STEROID THERAPY I/V Methyl Prednisolone 250 mg 6 hrly for 3 days foll by Oral Prednisolone 1mg/kg/day for 11 days Taper off over 3 days Follow up : Follow up 31 Mar 08 – Vision improved drastically to 4/60 LE 01 April 08 – Vision improved further to 6/36 03 April 08 – Vision improved to 6/18 05 April 08 – Vision improved to 6/9p TODAY O7 APRIL VISION IS 6/6P Conclusion : Conclusion Optic Neuritis is a serious condition which if not treated immediately can lead to permanent visual loss. ANY PATIENT COMPLAINING OF SUDDEN DETERIORATION OF VISION should be critically examined , thoroughly investigated and promptly managed.