logging in or signing up Examination of the Spine ramitgupta Download Post to : URL : Related Presentations : Share Add to Flag Embed Email Send to Blogs and Networks Add to Channel Uploaded from authorPOINT lite Insert YouTube videos in PowerPont slides with aS Desktop Copy embed code: Embed: Flash iPad Dynamic Copy Does not support media & animations Automatically changes to Flash or non-Flash embed WordPress Embed Customize Embed URL: Copy Thumbnail: Copy The presentation is successfully added In Your Favorites. Views: 400 Category: Education License: All Rights Reserved Like it (0) Dislike it (0) Added: November 15, 2011 This Presentation is Public Favorites: 0 Presentation Description No description available. Comments Posting comment... Premium member Presentation Transcript History Taking: 1 History TakingPersonal History: 2 Name Age Sex Occupation Residence Marital state, number of children Menstrual history in some cases Special habits of medical importance (e.g. smoking: number of cigarettes & duration) Social history Personal HistoryComplaint:: 3 In the patient’s own words & duration Complaint:History of the present illness: 4 Symptoms are analyzed in relation to each other and chronologically. Onset : Sudden ,acute or gradual. Course: - Progressive - Stationary - Regressive - Fluctuating - Responce to Tx Negative information in some cases may be important e.g. central chest pain not related to effort Review of other systems History of the present illnessPast history:: 5 Previous illness Drugs Pregnancies. Family history: Similar disease DM Hypertension Allergy Trauma Surgery Blood transfusion Travel abroad Radiotherapy Past history:Example of a symptom’s analysis: 6 Pain : ask about 1-Site: Midline pain arise from single structure ( heart -liver- pancrease -GIT). Localized or diffuse Diaphragmatic pleura --> tip of shoulder Myocardial ischemia --> retrosternal, Lt arm, Lt shoulder 3-Character of pain: aching, colicky, stabbing, burning……. 2- Radiation : Example of a symptom’s analysisPain (cont .): 7 4-Severity of pain 5-Duration: e.g. - Trigemimd neuralgia ( seconds) - Intestinal colic (minutes) 6-Frequency & Periodicity 7-Time of occurence: e.g. Morning headache in migraine & HTN After rising in frontal sinusits. At the end of the day in tension headache Pain (cont .)Pain (cont .): 8 8- Aggravating factors : e.g. swallowing in esophagitis 9- Relieving factors: e.g. stop walking in ischemic pain 10-Associated phenomena: e.g. marked swaeting in cardiac pain Pain (cont .)General Examination: 9 General ExaminationRoutine Data or Vital Signs :: 10 Temperature Pulse Blood pressure Respiratory rate Routine Data or Vital Signs :Radial Artery: 11 Radial ArteryPulse Examination: 12 Pulse ExaminationSphygmomanometer cuff: 13 Sphygmomanometer cuffLength of the cuff: 14 Length of the cuffStethoscope: 15 StethoscopeBrachial Artery: 16 Brachial ArteryBP measurement: 17 BP measurementTemperature: 18 Normal: 36 .5 - 37.2 0 c -Diurnal variation -Age -Menstrual cycle variation Fever: T> 37.4 0 c -Infection -Tissue injury TemperatureHyperpyrexia:T> 41.50c: 19 Neoplastic causes Collagen diseases Drugs Endocrine causes CNS causes Hyperpyrexia :T> 41.5 0 cHypothermia:: 20 Shock. Hypothyroidism Panhypopituitarism Starvation Damage to anterior hypothalamus Old age Drugs e.g. phenothiazine Hypothermia:Types of fever: 21 Types of fever Continuous Fever: Day & night changes < 1 0 C -Pneumonia -Meningitis Relapsing Fever: Brucellosis- spirochaetal relapsing fever- Bel Epstein fever (Hodgkin’s disease)- charcoat fever (biliary obstruction) Intermittent Fever: reaches base line during day - Malaria Remittent Fever : variation > 1 0 C -Septic conditionsColor changes: 22 Color changes Normal complexion depends on:- Thickness of skin Edema and Myxoedema pallor Vascularity , Hb Reduced Hg > 5 gm cyanosis Presence of pigments . Bilirubin ++ --> jaundice Caroten causes yellowish discolorationPallor: 23 Pallor Look for mucous membrane in inner aspect of lips Hb < 6 gm/dl --> pale palmar creases Causes of pallor: Anemia Anxiety Shock EdemaCyanosis: 24 Bluish discoloration -Central ~~~ lateral aspect of under surface of the tongue (warm hands) -Peripheral ~~~ extremities (cold hands) D.D of central cyanosis: ++ methemoglobin N.B red cyanosis is due to CO poisoning CyanosisCyanosis: 25 CyanosisBody Built1: 26 Body Built (weight & Hight) BMI = BW (kg) / Ht (m 2 ) Normal Over weight Obese Morbid Obesity 20-25 25-30 30-40 >40 Body Built 1Body Built2: 27 Factors affecting the body built : Racial Familial Genetic Endocrine Malnutrition in young age Child hood disease. Body Built Body Built 2Decubitus : : 28 Rigid dorsal decubitus Lateral decubitus Coild up Opisthotonus Orthopnea prayers posture Squatting Tri Pod position Decubitus :Tri Pod position: 29 Patient with emphysema bending over in Tri-Pod Position Tri Pod positionExamination of the HEAD & FACE: 30 Examination of the HEAD & FACEHEAD & FACE: 31 Head : Face : Size Shape Localized swelling Expression Edema and swelling Complexion Color change ( pallor - cyanosis - jaundice) Individual organs Asymmetry Malar flush ------ HEAD & FACEFacial swelling causing asymmetry: 32 Facial swelling Facial swelling causing asymmetryFacial swelling:Rt periorbital: 33 Facial swelling:Rt periorbitalNormal Eye: 34 Normal EyeJaundice: 35 JaundiceJaundice2: 36 Jaundice2Eye Lids: 37 Edema Xanthelasma Dark ring Ptosis 3rd nerve paralysis - Horner’s syndrome Myathenia gravis Congenital retraction Thyrotoxicosis Eye LidsEdema of the eye lids: 38 Edema of the eye lidsEye ball: 39 Exophthalmus: Congenital Local condition Cavernous sinus A.V aneurysm Thyrotoxicosis Enophthalmus: dehydration ----- Eye ballPupils: 40 Pupils : size – equality - light reaction Small pinpoint pupils Mid position fixed pupil: (4-6mm) slightly dilated with no light reaction Mid brain damage Bilateral small pupils Unilateral ( 1- 2.5 nm ) < 1mm Sympath. Damage Hypothalamic Metabolic pontine Hge Drugs: morphine, heroin , narcoti Horner syndrome PupilsHorner syndrome: 41 Horner syndromePupils2: 42 “Large” dilated pupils anoxia Bilaterally dilated pupils (fixed): drugs (atropine - phenothiazine- tricyclic antidepressants) (Reactive) Unilaterally dilated pupil ( Fixed dilated) Oculomotor paralysis Temporal lobe herniation ---Mid brain . Cocaine Amphetamine, LSD (Sympath. Agonists ) Pupils 2Conjunctiva: 43 Hemorrhage Subconjunctivl haemorrage with severe cough Hypertension Septicaemia Bleeding tendency Conjunctiva Chemosis: edemaConjunctivitis: 44 ConjunctivitisSclera: 45 Sclera Scleritis EpiscleritisScleritis: 46 ScleritisNodular Episcleritis in a patient with CD: 47 Nodular Episcleritis in a patient with CDCornea: 48 Nose Ear Cornea Tophi Discoloration Cyanosis Ochronosis DischrgeParotid glands: 49 Unilateral enlargement as in acute parotitis Bilateral enlargement as in Sjogren Syndrome Parotid glandsUnilaterally enlarged parotid: 50 Unilaterally enlarged parotidMouth: Lips: 51 Color Angular stomatitis Chelitis Hypertrophy Herpes labialis Acromegaly Telangiectasia Myxoedema Trauma Angioedema Mouth: LipsBreath: 52 Fetid breath (Fetor oris) Breath Alcohol Acetone : D.K.A Ammonia ---> uraemia Fetor hepaticus Local oral condition pyorrhea Suppurative lung syndrome Pyloric obstructionMouth : Dryness and increased salivation: 53 Dehydration Mouth breathing Anxiety Drugs, e.g. anticholinergics Sj ö gren syndrome Causes of Dry Mouth Ptyalism: increased salivation Mouth : Dryness and increased salivationNormal oral cavity: 54 Normal oral cavityLeft peritonsillar abscess: 55 Left peritonsillar abscessTeeth: 56 Loose teeth: - D.M - Hyperparathyroidism Wide spaced teeth: acromegaly Discoloration: Teeth Tobacco Poor oral hygiene Flourosis xxxxxTooth abscess: 57 Tooth Abscess Tooth abscessGums: 58 Pyorrhea Bleeding as in: Gums Hypertrophy as in: Addison Heavy metal ( lead and bisthmus) poisoning Drugs like epanutin Monocytic leukemia Pigmentation as in: Chronic liver disease ThrombocytopeniaTongue1: 59 color Tongue 1 pale Bright red ( firy tongue- pellagra) Atrophic glossitis ( iron , B12 , riboflavin - pellagra) Black --> iron mixture coated --> dehydration Strawberry tongue scarlet feve Leukoplakia: precancerous Pigment : Addison’s diseaseTongue2: 60 Dry tongue (see causes of dry mouth) Scrotal tongue (mongolism) Tremors Tongue 2 Large tongue (macroglossia) as in acromegaly Anxiety Smoking Parkinsonism Chronic AlcoholismBuccal mucosa1: 61 Buccal mucosa 1 Stomatitis (Red, swollen& tender) Catarrhal Ulcerative Monilia Aphthous Vincent’s angina Pigmentation Dark skinned Addison disease Intestinal polyposis Arsenic HemochromatosisBuccal mucosa2: 62 Buccal mucosa 2 Enanthema : Koplik’s spots Petechial Hge: Infective endocarditis - leukaemia Palate: Tonsils Deformity Paralysis VesiclesNeck: 63 Neck Insepction Palpation Lymph Nodes Salivary glands Thyroid other swellings Deformity Asymmetry Position Limitation of movement PulsationsThyroid gland:anatomy: 64 Isthmus Thyroid gland:anatomyPalpation of the thyroid gland:posterior approach: 65 Palpation of the thyroid gland:posterior approachPalpation of the thyroid gland:anterior approach: 66 Palpation of the thyroid gland: anterior approachNeck veins: 67 Jugular Vein Carotid Artery No pulsations palpable. Palpable pulsations. Pulsations obliterated by pressure above the clavicle. Pulsations not obliterated by pressure above the clavicle. Level of pulse wave decreased on inspiration; increased on expiration. No effects of respiration on pulse. Usually two pulsations per systole (x and y descents). One pulsation per systole. Prominent descents. Descents not prominent. Pulsations sometimes more prominent with abnominal pressure. No effect of abdominal pressure on pulsations Neck veinsCervical lymph nodes: 68 Cervical lymph nodesExamination of Axillae: 69 Examination of Axillae Lymph nodes Any swellingExamination of the axilla1: 70 Examination of the axilla 1Axillary lymphadenopathy: 71 Axillary lymphadenopathyExamination of the axilla2: 72 Examination of the axilla 2Examination of the Breast : 73 Examination of the Breast Gynecomastia: Physialogicl Drugs Liver cell failure Klinefelter Testiculr atrophy and tumors Adrenal tumors Estrogen Spironolactone ChloropromazineEpitrochlear LN: 74 Epitrochlear LNExamination of the Upper Limbs:Edema: 75 Examination of the Upper Limbs:EdemaDVT of the right arm: 76 DVT of the right armCellulitis of the upper limb: 77 Examination of upper limbs:Cellulitis Cellulitis of the upper limbCellulitis of the upper limb 2: 78 Cellulitis of the upper limb 2Examination of Hands: 79 Examination of Hands Shape & Size Wasting of the muscles Signs of peripheral vascular disease Staining of nails: as in heavy cig smokers White nails uraemia - liver cell failure Splinter Hg S.A.B.E See also joint examinationAcromegaly: 80 AcromegalyPeripheral Vascular Disease of the UL: 81 Peripheral Vascular Disease of the ULWasting of the thenar eminance: 82 Wasting of the thenar eminanceNicotine staining: 83 Nicotine stainingWrist: Ganglion: 84 Wrist: GanglionSkin1: 85 Skin 1 Pigmentation Texture Elasticity Thickness Striae EruptionSkin2: 86 Skin 2 Pigmentation Generalized Locaalized Addison’s disease Pellagra Hemochromatosis Ochronosis Arsenic Cloasma Sun burn Post- inflammatory X-ray Neurofibromatosis Leucoderma: -Vitiligo -leprosy - SLE - Pityriasis versicolorSkin3: 87 Skin 3 2-Texture: Dryness Sweating Dehydration Myxedema Anxiety Thyrotoxicosis Respiratory failure Hyroglycaemia ToxemiaSkin4: 88 3.Elasticity : cutis loxa - old age - progeria 4.Thickness : -Acromegaly - Elephantiasis - Occupational 5.Striae: Pregnancy Obesity Cushing’s syndrome Skin 4Skin5: 89 6-Eruption: Skin 5 Distribution: distribution of sensory nerve HZ Arrangement: Morphology: Linear lymphangitis Annular psoriasis Serpiginous Syphilis Irregular urticria Monomorphic Pleomorphic Type: Macule- papule- nodule- plaque- vesicles- bullae- pustule - wheals - scales - crust – erosion - fissure- ulcers – scar - atrophy - sclerosis.Skin6: 90 Dilated Blood vessels Skin 6 Petechie 1-2 mm (Hess test) Purpura: e.g. thrombocytopenia , Senile purpura Ecchymosis > 5 mm Collaterals Telangiectsia. Haemangioma Vascular spiders Campbell de Morgan spotsTelangiectasia: 91 TelangiectasiaSkin-Hair: 92 Fall of hair : Look for Distribution: {head, face, axillae, and pubis) Skin-Hair Hirsutism: excessive growth of body hair of a female Idiopathic Racial Endocrine: Cushing- adrenogenital syndrome- polycystic ovary. Endocrine: Myxoedem - Addison’s disease - Sheehan’s syndrome- Eunchiadism Infection Localized fall of hair: Alopecia areataLymph nodes1: 93 Lymph nodes 1 Group {cervical (superfacial and deep) , scalene (Lt virchow), axillary, epitrochlear, inguinal }. Size Consistency Tenderness Matting Mobility Relation to surrounding structures.Lymph nodes2: 94 Lymph nodes 2 Lymphadenopathy: Localized : Acute lymphadenitis T.B Hodgkin’ disease Generalized: Viral : glandulr fever Leukemia . Lymphoma Syphilis SarcoidosisExamination of the Lower Limbs: 95 Examination of the Lower LimbsPeripheral pulsation :Dorsalis pedis: 96 Peripheral pulsation :Dorsalis pedisPeripheral pulsations :Dorsalis pedis 2: 97 Peripheral puls ations :Dorsalis pedis 2Peripheral pulsations :post tibial artery: 98 Peripheral pulsations :post tibial arteryPeripheral pulsations :posterior tibial artery2: 99 Peripheral pulsations : posterior tibial artery 2Popliteal artery: 100 Popliteal arteryAcute vascular insufficiency:mottled appearance: 101 Acute vascular insufficiency: mottled appearanceChronic arterial insufficiency: 102 Chronic arterial insufficiencyDigital gangrene: 103 Digital gangreneChronic Venous Insufficiency: 104 Chronic Venous InsufficiencyNeuropathic ulcer in a diabetic patient: 105 Neuropathic ulcer in a diabetic patientLower limbs: edema1: 106 Inspection Pressure over bony prominance for 5 to 30 sec just behind and below medial malleoli and sacrum For soft tissue edema: Lower limbs: edema 1 pinching dimpling of skin ( Peou’d ‘orange) press with the stethoscopeLower limbs:Edema2: 107 Edema may be: Lower limbs:Edema 2 Hard as in chronic. lymphatic obstruction. Soft Causes of Soft Edema: Localized edema: Inflammtory (hot, red, tender)- Angioneurotic - DVT- Paralysis --> paralyzed side Generlized edema: Renal Cardiac Nutritional HepaticLL edema: 108 LL edemaPitting edema of the lower limb: 109 Pitting edema of the lower limbErythema nodosum: 110 Erythema nodosumOnychomycosis: 111 OnychomycosisGenitalia and Joints: 112 Genitalia Joints : Swelling Deformity Overlying skin Tenderness Atrophy of muscles Limitation of movement Hypermobility Eruption Genitalia and JointsRheumatoid arthritis: 113 Rheumatoid arthritisRA: boutonniere finger: 114 RA: boutonniere fingerRheumatoid arthritis 2: 115 Rheumatoid arthritis 2Heberdens nodes: 116 Heberdens nodes Heberdens nodesGout: MCP joints: 117 Gout: MCP jointsLeprosy: 118 Leprosy You do not have the permission to view this presentation. In order to view it, please contact the author of the presentation.
Examination of the Spine ramitgupta Download Post to : URL : Related Presentations : Share Add to Flag Embed Email Send to Blogs and Networks Add to Channel Uploaded from authorPOINT lite Insert YouTube videos in PowerPont slides with aS Desktop Copy embed code: Embed: Flash iPad Dynamic Copy Does not support media & animations Automatically changes to Flash or non-Flash embed WordPress Embed Customize Embed URL: Copy Thumbnail: Copy The presentation is successfully added In Your Favorites. Views: 400 Category: Education License: All Rights Reserved Like it (0) Dislike it (0) Added: November 15, 2011 This Presentation is Public Favorites: 0 Presentation Description No description available. Comments Posting comment... Premium member Presentation Transcript History Taking: 1 History TakingPersonal History: 2 Name Age Sex Occupation Residence Marital state, number of children Menstrual history in some cases Special habits of medical importance (e.g. smoking: number of cigarettes & duration) Social history Personal HistoryComplaint:: 3 In the patient’s own words & duration Complaint:History of the present illness: 4 Symptoms are analyzed in relation to each other and chronologically. Onset : Sudden ,acute or gradual. Course: - Progressive - Stationary - Regressive - Fluctuating - Responce to Tx Negative information in some cases may be important e.g. central chest pain not related to effort Review of other systems History of the present illnessPast history:: 5 Previous illness Drugs Pregnancies. Family history: Similar disease DM Hypertension Allergy Trauma Surgery Blood transfusion Travel abroad Radiotherapy Past history:Example of a symptom’s analysis: 6 Pain : ask about 1-Site: Midline pain arise from single structure ( heart -liver- pancrease -GIT). Localized or diffuse Diaphragmatic pleura --> tip of shoulder Myocardial ischemia --> retrosternal, Lt arm, Lt shoulder 3-Character of pain: aching, colicky, stabbing, burning……. 2- Radiation : Example of a symptom’s analysisPain (cont .): 7 4-Severity of pain 5-Duration: e.g. - Trigemimd neuralgia ( seconds) - Intestinal colic (minutes) 6-Frequency & Periodicity 7-Time of occurence: e.g. Morning headache in migraine & HTN After rising in frontal sinusits. At the end of the day in tension headache Pain (cont .)Pain (cont .): 8 8- Aggravating factors : e.g. swallowing in esophagitis 9- Relieving factors: e.g. stop walking in ischemic pain 10-Associated phenomena: e.g. marked swaeting in cardiac pain Pain (cont .)General Examination: 9 General ExaminationRoutine Data or Vital Signs :: 10 Temperature Pulse Blood pressure Respiratory rate Routine Data or Vital Signs :Radial Artery: 11 Radial ArteryPulse Examination: 12 Pulse ExaminationSphygmomanometer cuff: 13 Sphygmomanometer cuffLength of the cuff: 14 Length of the cuffStethoscope: 15 StethoscopeBrachial Artery: 16 Brachial ArteryBP measurement: 17 BP measurementTemperature: 18 Normal: 36 .5 - 37.2 0 c -Diurnal variation -Age -Menstrual cycle variation Fever: T> 37.4 0 c -Infection -Tissue injury TemperatureHyperpyrexia:T> 41.50c: 19 Neoplastic causes Collagen diseases Drugs Endocrine causes CNS causes Hyperpyrexia :T> 41.5 0 cHypothermia:: 20 Shock. Hypothyroidism Panhypopituitarism Starvation Damage to anterior hypothalamus Old age Drugs e.g. phenothiazine Hypothermia:Types of fever: 21 Types of fever Continuous Fever: Day & night changes < 1 0 C -Pneumonia -Meningitis Relapsing Fever: Brucellosis- spirochaetal relapsing fever- Bel Epstein fever (Hodgkin’s disease)- charcoat fever (biliary obstruction) Intermittent Fever: reaches base line during day - Malaria Remittent Fever : variation > 1 0 C -Septic conditionsColor changes: 22 Color changes Normal complexion depends on:- Thickness of skin Edema and Myxoedema pallor Vascularity , Hb Reduced Hg > 5 gm cyanosis Presence of pigments . Bilirubin ++ --> jaundice Caroten causes yellowish discolorationPallor: 23 Pallor Look for mucous membrane in inner aspect of lips Hb < 6 gm/dl --> pale palmar creases Causes of pallor: Anemia Anxiety Shock EdemaCyanosis: 24 Bluish discoloration -Central ~~~ lateral aspect of under surface of the tongue (warm hands) -Peripheral ~~~ extremities (cold hands) D.D of central cyanosis: ++ methemoglobin N.B red cyanosis is due to CO poisoning CyanosisCyanosis: 25 CyanosisBody Built1: 26 Body Built (weight & Hight) BMI = BW (kg) / Ht (m 2 ) Normal Over weight Obese Morbid Obesity 20-25 25-30 30-40 >40 Body Built 1Body Built2: 27 Factors affecting the body built : Racial Familial Genetic Endocrine Malnutrition in young age Child hood disease. Body Built Body Built 2Decubitus : : 28 Rigid dorsal decubitus Lateral decubitus Coild up Opisthotonus Orthopnea prayers posture Squatting Tri Pod position Decubitus :Tri Pod position: 29 Patient with emphysema bending over in Tri-Pod Position Tri Pod positionExamination of the HEAD & FACE: 30 Examination of the HEAD & FACEHEAD & FACE: 31 Head : Face : Size Shape Localized swelling Expression Edema and swelling Complexion Color change ( pallor - cyanosis - jaundice) Individual organs Asymmetry Malar flush ------ HEAD & FACEFacial swelling causing asymmetry: 32 Facial swelling Facial swelling causing asymmetryFacial swelling:Rt periorbital: 33 Facial swelling:Rt periorbitalNormal Eye: 34 Normal EyeJaundice: 35 JaundiceJaundice2: 36 Jaundice2Eye Lids: 37 Edema Xanthelasma Dark ring Ptosis 3rd nerve paralysis - Horner’s syndrome Myathenia gravis Congenital retraction Thyrotoxicosis Eye LidsEdema of the eye lids: 38 Edema of the eye lidsEye ball: 39 Exophthalmus: Congenital Local condition Cavernous sinus A.V aneurysm Thyrotoxicosis Enophthalmus: dehydration ----- Eye ballPupils: 40 Pupils : size – equality - light reaction Small pinpoint pupils Mid position fixed pupil: (4-6mm) slightly dilated with no light reaction Mid brain damage Bilateral small pupils Unilateral ( 1- 2.5 nm ) < 1mm Sympath. Damage Hypothalamic Metabolic pontine Hge Drugs: morphine, heroin , narcoti Horner syndrome PupilsHorner syndrome: 41 Horner syndromePupils2: 42 “Large” dilated pupils anoxia Bilaterally dilated pupils (fixed): drugs (atropine - phenothiazine- tricyclic antidepressants) (Reactive) Unilaterally dilated pupil ( Fixed dilated) Oculomotor paralysis Temporal lobe herniation ---Mid brain . Cocaine Amphetamine, LSD (Sympath. Agonists ) Pupils 2Conjunctiva: 43 Hemorrhage Subconjunctivl haemorrage with severe cough Hypertension Septicaemia Bleeding tendency Conjunctiva Chemosis: edemaConjunctivitis: 44 ConjunctivitisSclera: 45 Sclera Scleritis EpiscleritisScleritis: 46 ScleritisNodular Episcleritis in a patient with CD: 47 Nodular Episcleritis in a patient with CDCornea: 48 Nose Ear Cornea Tophi Discoloration Cyanosis Ochronosis DischrgeParotid glands: 49 Unilateral enlargement as in acute parotitis Bilateral enlargement as in Sjogren Syndrome Parotid glandsUnilaterally enlarged parotid: 50 Unilaterally enlarged parotidMouth: Lips: 51 Color Angular stomatitis Chelitis Hypertrophy Herpes labialis Acromegaly Telangiectasia Myxoedema Trauma Angioedema Mouth: LipsBreath: 52 Fetid breath (Fetor oris) Breath Alcohol Acetone : D.K.A Ammonia ---> uraemia Fetor hepaticus Local oral condition pyorrhea Suppurative lung syndrome Pyloric obstructionMouth : Dryness and increased salivation: 53 Dehydration Mouth breathing Anxiety Drugs, e.g. anticholinergics Sj ö gren syndrome Causes of Dry Mouth Ptyalism: increased salivation Mouth : Dryness and increased salivationNormal oral cavity: 54 Normal oral cavityLeft peritonsillar abscess: 55 Left peritonsillar abscessTeeth: 56 Loose teeth: - D.M - Hyperparathyroidism Wide spaced teeth: acromegaly Discoloration: Teeth Tobacco Poor oral hygiene Flourosis xxxxxTooth abscess: 57 Tooth Abscess Tooth abscessGums: 58 Pyorrhea Bleeding as in: Gums Hypertrophy as in: Addison Heavy metal ( lead and bisthmus) poisoning Drugs like epanutin Monocytic leukemia Pigmentation as in: Chronic liver disease ThrombocytopeniaTongue1: 59 color Tongue 1 pale Bright red ( firy tongue- pellagra) Atrophic glossitis ( iron , B12 , riboflavin - pellagra) Black --> iron mixture coated --> dehydration Strawberry tongue scarlet feve Leukoplakia: precancerous Pigment : Addison’s diseaseTongue2: 60 Dry tongue (see causes of dry mouth) Scrotal tongue (mongolism) Tremors Tongue 2 Large tongue (macroglossia) as in acromegaly Anxiety Smoking Parkinsonism Chronic AlcoholismBuccal mucosa1: 61 Buccal mucosa 1 Stomatitis (Red, swollen& tender) Catarrhal Ulcerative Monilia Aphthous Vincent’s angina Pigmentation Dark skinned Addison disease Intestinal polyposis Arsenic HemochromatosisBuccal mucosa2: 62 Buccal mucosa 2 Enanthema : Koplik’s spots Petechial Hge: Infective endocarditis - leukaemia Palate: Tonsils Deformity Paralysis VesiclesNeck: 63 Neck Insepction Palpation Lymph Nodes Salivary glands Thyroid other swellings Deformity Asymmetry Position Limitation of movement PulsationsThyroid gland:anatomy: 64 Isthmus Thyroid gland:anatomyPalpation of the thyroid gland:posterior approach: 65 Palpation of the thyroid gland:posterior approachPalpation of the thyroid gland:anterior approach: 66 Palpation of the thyroid gland: anterior approachNeck veins: 67 Jugular Vein Carotid Artery No pulsations palpable. Palpable pulsations. Pulsations obliterated by pressure above the clavicle. Pulsations not obliterated by pressure above the clavicle. Level of pulse wave decreased on inspiration; increased on expiration. No effects of respiration on pulse. Usually two pulsations per systole (x and y descents). One pulsation per systole. Prominent descents. Descents not prominent. Pulsations sometimes more prominent with abnominal pressure. No effect of abdominal pressure on pulsations Neck veinsCervical lymph nodes: 68 Cervical lymph nodesExamination of Axillae: 69 Examination of Axillae Lymph nodes Any swellingExamination of the axilla1: 70 Examination of the axilla 1Axillary lymphadenopathy: 71 Axillary lymphadenopathyExamination of the axilla2: 72 Examination of the axilla 2Examination of the Breast : 73 Examination of the Breast Gynecomastia: Physialogicl Drugs Liver cell failure Klinefelter Testiculr atrophy and tumors Adrenal tumors Estrogen Spironolactone ChloropromazineEpitrochlear LN: 74 Epitrochlear LNExamination of the Upper Limbs:Edema: 75 Examination of the Upper Limbs:EdemaDVT of the right arm: 76 DVT of the right armCellulitis of the upper limb: 77 Examination of upper limbs:Cellulitis Cellulitis of the upper limbCellulitis of the upper limb 2: 78 Cellulitis of the upper limb 2Examination of Hands: 79 Examination of Hands Shape & Size Wasting of the muscles Signs of peripheral vascular disease Staining of nails: as in heavy cig smokers White nails uraemia - liver cell failure Splinter Hg S.A.B.E See also joint examinationAcromegaly: 80 AcromegalyPeripheral Vascular Disease of the UL: 81 Peripheral Vascular Disease of the ULWasting of the thenar eminance: 82 Wasting of the thenar eminanceNicotine staining: 83 Nicotine stainingWrist: Ganglion: 84 Wrist: GanglionSkin1: 85 Skin 1 Pigmentation Texture Elasticity Thickness Striae EruptionSkin2: 86 Skin 2 Pigmentation Generalized Locaalized Addison’s disease Pellagra Hemochromatosis Ochronosis Arsenic Cloasma Sun burn Post- inflammatory X-ray Neurofibromatosis Leucoderma: -Vitiligo -leprosy - SLE - Pityriasis versicolorSkin3: 87 Skin 3 2-Texture: Dryness Sweating Dehydration Myxedema Anxiety Thyrotoxicosis Respiratory failure Hyroglycaemia ToxemiaSkin4: 88 3.Elasticity : cutis loxa - old age - progeria 4.Thickness : -Acromegaly - Elephantiasis - Occupational 5.Striae: Pregnancy Obesity Cushing’s syndrome Skin 4Skin5: 89 6-Eruption: Skin 5 Distribution: distribution of sensory nerve HZ Arrangement: Morphology: Linear lymphangitis Annular psoriasis Serpiginous Syphilis Irregular urticria Monomorphic Pleomorphic Type: Macule- papule- nodule- plaque- vesicles- bullae- pustule - wheals - scales - crust – erosion - fissure- ulcers – scar - atrophy - sclerosis.Skin6: 90 Dilated Blood vessels Skin 6 Petechie 1-2 mm (Hess test) Purpura: e.g. thrombocytopenia , Senile purpura Ecchymosis > 5 mm Collaterals Telangiectsia. Haemangioma Vascular spiders Campbell de Morgan spotsTelangiectasia: 91 TelangiectasiaSkin-Hair: 92 Fall of hair : Look for Distribution: {head, face, axillae, and pubis) Skin-Hair Hirsutism: excessive growth of body hair of a female Idiopathic Racial Endocrine: Cushing- adrenogenital syndrome- polycystic ovary. Endocrine: Myxoedem - Addison’s disease - Sheehan’s syndrome- Eunchiadism Infection Localized fall of hair: Alopecia areataLymph nodes1: 93 Lymph nodes 1 Group {cervical (superfacial and deep) , scalene (Lt virchow), axillary, epitrochlear, inguinal }. Size Consistency Tenderness Matting Mobility Relation to surrounding structures.Lymph nodes2: 94 Lymph nodes 2 Lymphadenopathy: Localized : Acute lymphadenitis T.B Hodgkin’ disease Generalized: Viral : glandulr fever Leukemia . Lymphoma Syphilis SarcoidosisExamination of the Lower Limbs: 95 Examination of the Lower LimbsPeripheral pulsation :Dorsalis pedis: 96 Peripheral pulsation :Dorsalis pedisPeripheral pulsations :Dorsalis pedis 2: 97 Peripheral puls ations :Dorsalis pedis 2Peripheral pulsations :post tibial artery: 98 Peripheral pulsations :post tibial arteryPeripheral pulsations :posterior tibial artery2: 99 Peripheral pulsations : posterior tibial artery 2Popliteal artery: 100 Popliteal arteryAcute vascular insufficiency:mottled appearance: 101 Acute vascular insufficiency: mottled appearanceChronic arterial insufficiency: 102 Chronic arterial insufficiencyDigital gangrene: 103 Digital gangreneChronic Venous Insufficiency: 104 Chronic Venous InsufficiencyNeuropathic ulcer in a diabetic patient: 105 Neuropathic ulcer in a diabetic patientLower limbs: edema1: 106 Inspection Pressure over bony prominance for 5 to 30 sec just behind and below medial malleoli and sacrum For soft tissue edema: Lower limbs: edema 1 pinching dimpling of skin ( Peou’d ‘orange) press with the stethoscopeLower limbs:Edema2: 107 Edema may be: Lower limbs:Edema 2 Hard as in chronic. lymphatic obstruction. Soft Causes of Soft Edema: Localized edema: Inflammtory (hot, red, tender)- Angioneurotic - DVT- Paralysis --> paralyzed side Generlized edema: Renal Cardiac Nutritional HepaticLL edema: 108 LL edemaPitting edema of the lower limb: 109 Pitting edema of the lower limbErythema nodosum: 110 Erythema nodosumOnychomycosis: 111 OnychomycosisGenitalia and Joints: 112 Genitalia Joints : Swelling Deformity Overlying skin Tenderness Atrophy of muscles Limitation of movement Hypermobility Eruption Genitalia and JointsRheumatoid arthritis: 113 Rheumatoid arthritisRA: boutonniere finger: 114 RA: boutonniere fingerRheumatoid arthritis 2: 115 Rheumatoid arthritis 2Heberdens nodes: 116 Heberdens nodes Heberdens nodesGout: MCP joints: 117 Gout: MCP jointsLeprosy: 118 Leprosy