Myocardial Infarction Imaging

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Myocardial Infarction Imaging

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Added: July 31, 2008 This Presentation is Public 
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Myocardial infarction(PYP imaging) :Myocardial infarction(PYP imaging)


Anatomy :Anatomy Myocardial Infarction Left Ventricle Areas affected Anterior wall and septum Inferior wall and posterior third of septum Right ventricle involved in 25% to 50 of pt with inferior wall infarction


Classification of infarction :Classification of infarction Thickness of necrosis Transmural or Q wave- full thickness myocardial wall Nontransmural, non-Q wave- less than full thickness of wall Subendocardial infarction- less than one-half thickness of wall.


Slide 4:Coronary thrombosis Present in half of infarcts Intramural hemorrhage 40% Most frequent arteries: LAD (56%) Right coronary (25%) Circumflex (14%) Tans mural infarction Coronary artery spasm Some primary cause of infarction Causes for Infarction


Physiology :Physiology Myocardial Infarction Intramural infarctions Reduced blood flow to center of infarction= endocardium


Slide 6:Technitium 99m pyrophosphate Determinants of Uptake in infarction. Blood Flow Tc 99m PYP must reach damaged tissue to be taken up. Highest concentration of 99mTc pyrophosphate occurs when local blood flow reduced by 20% to 40% Flow reduces, concentration falls in regions of severely reduced flow Concentration of pyrophosphate greater in epicardial than endocardial segments PYP ratio of 18 to 20 infarcted tissue/ normal myocardium possible.


Slide 7:Extraction Extraction fraction Total amount tracer extracted decreases at low flow. Calcium influx Key role in T99m PYP binding acute infarction Intracellular calcium phosphate and is present in 3 forms in cell: Amorphous – uniformly Crystalline Hydroxyapatite- mitochondria PYP binds to both amorphous and hydroxyapatite deposits 99mTc PYP concentration parallels calcium phosphate concentration.


Slide 8:Necrotic Tissue Pyrophosphate Acutely necrotic myocardium Other Diseases unstable angina, multifocal lesions of necrosis and myocytolysis Uptake in ventricular aneurysm


Timing :Timing Earliest uptake 4 hours after coronary artery occulsion Peak uptake @ 48 hours Diminish over next 5 to 7 days Time course varies depending on: Size of infarction Extension of infarction


Slide 10:CAD


Slide 11:CAD


Slide 16:MI


Myocardial Infarction :Myocardial Infarction


Slide 18:PYP