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Premium member Presentation Transcript SKIN CANCERS : SKIN CANCERS Many photos and information courtesy of Dr Cliff Rosendall Dr Alan Cameron SCCANZ and Dermoscopy atlas What is a skin cancer : What is a skin cancer It is a mutation in the skin with more aggressive growth potential and lacking general structural restraints. Due to numerous factors including sun damage Family history (genetics) Exposure to certain chemicals and foods Chemicals and foods : Chemicals and foods Arsenic – sheep dip ,pottery glazing, wells Tars Dry cleaning liquids Asbestos Fibreglass Chemotherapy Smoking Solarium use (esp. under 35 yrs old) Risk factors to identify : Risk factors to identify Fair skin blue eyes(type 1, 2 Fitzpatrick skin) Previous melanoma/skin cancer and family history Occupational exposure (sun or chemicals) Immune suppressed – organ transplants Multiple moles Previous chemical exposure Sun burned history especially when young Illnesses- leukaemia ,rheumatoid arthritis Prevalence : Prevalence Skin cancer is the most common cancer in the world affecting up to 40% of all north Queenslanders over 50 years old 1 in 200 Australians will develop melanoma (I think this is very conservative) 3 Australians die each day from melanoma. 6 a day in the UK But remember : But remember It is also the easiest cancer to treat if diagnosed early -as it is visible and exposed –you only need to be able to recognise it and understand the risks that lie below the surface Slide 8: IF IN DOUBT ...REFER TO A SKIN CANCER DOCTOR because if you cut the surface off and leave the roots –it will spread deeper and become more aggressive Mechanism of formation : Mechanism of formation Slide 11: epidermis dermis Damage to skin External causes – sun etc Internal causes Hereditary- already there awaiting a “trigger” Slide 12: epidermis dermis Non invasive stage – contained in epidermis – IEC-intra epidermal squamous cell cancer Superficial basal cell cancer Melanoma in situ, Hutchinson's melanotic freckle Slide 13: epidermis dermis Invasive stage BCC SCC Invasive Melanoma WHAT TO LOOK OUT FOR : WHAT TO LOOK OUT FOR Slide 15: ANYTHING NEW OR WAS NOT THERE BEFORE Slide 18: ANY ODD OR DIFFERENT LESION THE UGLY DUCKLING SYNDROME ANY GROWING OR CHANGING LESION : ANY GROWING OR CHANGING LESION Anything bleeding itching or a lesion that keeps coming back : Anything bleeding itching or a lesion that keeps coming back Slide 25: Nodular bcc SIMPLE ABC RULE : SIMPLE ABC RULE A – asymetry B – border C – colour D – diameter E – elevation F – firmness G - growing Slide 27: AsymmetryAsymmetry can be assessed by comparing one half of the growth to the other half to determine if the halves are equal in size. -Unequal or asymmetric moles are suspicious. Symmetric (normal) Asymmetric Slide 28: REGULAR IRREGULAR Slide 29: Single colour Multiple colours Slide 30: DiameterAny mole that has a diameter larger than a pencil's eraser in size (> 6 mm) should be considered suspicious Slide 31: ElevationIf a mole is elevated, or raised from of the skin, it should be considered suspicious. THE MOST IMPORTANT TOOL IS THE DERMATOSCOPE : THE MOST IMPORTANT TOOL IS THE DERMATOSCOPE TAKE HOME MESSAGE : TAKE HOME MESSAGE IF YOU ARE GOING TO REMOVE ANY LESION BY CAUTERY, LASER, IPL, HYPHRECATION , SHAVING ,CRYOTHERAPY OR EXCISION GET A DERMATOSCOPE AND LEARN HOW TO DIFFERENTIATE BENIGN FROM MALIGNANT LESIONS Slide 34: REMEMBER IF YOU ONLY PARTLY DESTROY A LESION IT WILL CONTINUE TO GROW UNDER THE SKIN AND GET ANGRIER TYPES OF LESIONS : TYPES OF LESIONS BENIGN PRE-MALIGNANT MALIGNANT BENIGN : BENIGN CONGENITAL and INK SPOT NAEVI JUNCTIONAL AND DERMAL NAEVI AGE SPOTS – SEBORRHEIC KERATOSES FRECKLES SOLAR LENTIGO VASCULAR -HAEMANGIOMAS CAMPBELL DE MORGAN BLUE NAEVI Slide 38: CONGENITAL NAEVI CONGENITAL NAEVUS Slide 39: DERMAL NAEVUS Slide 40: SEBORRHEIC KERATOSIS Slide 41: Seborrheic keratoses Slide 42: freckles Slide 43: SOLAR LENTIGO Slide 44: HAEMANGIOMA Slide 45: BLUE NAEVUS PRE-MALIGNANT : PRE-MALIGNANT SUN SPOTS, ACTINIC KERATOSES, SOLAR KERATOSES DYSPLASTIC NAEVUS SPITZ NAEVUS Slide 47: SOLAR KERATOSES Slide 48: DYSPLASTIC NAEVI Slide 49: SPITZ NAEVI MALIGNANT : MALIGNANT NON MELANOCYTIC BCC SCC MELANOCYTIC MELANOMA BCC : BCC SUPERFICIAL NODULAR MICRONODULAR SCLEROSING BASI SQUAMOUS Slide 53: bcc SCC : SCC INTRA EPIDERMAL SCC – BOWENS, SCC IN SITU POORLY DIFFERENTIATED SCC MODERATELY DIFFERENTIATED SCC WELL DIFFERENTIATED SCC Kerato-akanthoma Slide 55: scc Slide 56: Kerato acanthoma MELANOMA : MELANOMA Hutchinsons melanotic freckle Lentigo maligna Melanoma in situ Superficial spreading melanoma Acral melanoma Subungual melanoma NODULAR MELANOMA LESIONS BY COLOUR : LESIONS BY COLOUR Black /blue Pink White Red Brown Yellow mixed Slide 67: BLACK OR BLUE MELANOMA BLUE NAEVUS BCC SPITZ NAEVUS STRANGULATED POLYP OR HAEMANGIOMA INK SPOT NAEVUS SEBORRHEIC KERATOSIS Slide 68: Pink MELANOMA BCC SCC KA VASCULAR LESIONS SOLAR KERATOSES SEBORRHEIC KERATOSES Slide 69: WHITE MELANOMA SCARS FUNGAL VITILIGO Slide 70: RED MELANOMA VASCULAR LESIONS KA BCC SCC Slide 71: BROWN MELANOMA NAEVI FRECKLES BCC SCC Slide 72: YELLOW ALL KERATINISING LESIONS IEC SCC KA SOLAR KERATOSES SEBORRHEIC KERATOSES Slide 73: MIXED MELANOMA BCC CONGENITAL NAEVUS DYSPLASTIC NAEVUS SEBORRHEIC KERATOSIS TREATMENT : TREATMENT EARLY DIAGNOSIS AND EXCISION WITH ADEQUATE MARGINS THANK YOU : THANK YOU You do not have the permission to view this presentation. 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