logging in or signing up Head and Brain Trauma aSGuest1066 Download Post to : URL : Related Presentations : Share Add to Flag Embed Email Send to Blogs and Networks Add to Channel Uploaded from authorPOINT lite Insert YouTube videos in PowerPont slides with aS Desktop Copy embed code: Embed: Flash iPad Dynamic Copy Does not support media & animations Automatically changes to Flash or non-Flash embed WordPress Embed Customize Embed URL: Copy Thumbnail: Copy The presentation is successfully added In Your Favorites. Views: 14317 Category: Education License: All Rights Reserved Like it (9) Dislike it (0) Added: October 15, 2008 This Presentation is Public Favorites: 16 Presentation Description No description available. Comments Posting comment... By: faizalahamed (51 month(s) ago) hello,this is a wonderful presentation to use for lecture. Saving..... Post Reply Close Saving..... 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Edit Comment Close Premium member Presentation Transcript Head & Brain Trauma : Head & Brain Trauma EMS Professions Temple College Head & Brain Trauma : Head & Brain Trauma ~ 4 million head injuries in US per year ~ 450, 000 require hospitalization Most are minor injuries Major head injury most common cause of trauma deaths in trauma centers (>50%) Head & Brain Trauma : Head & Brain Trauma Risk Groups Highest: Males 15-24 yrs of age Very young children: 6 mos to 2 yrs of age Young school age children Elderly Skull Anatomy Review : Skull Anatomy Review Cranium Double layer of solid bone which surrounds a spongy middle layer Frontal, occipital, temporal, parietal, mastoid Middle meningeal artery lies under temporal bone common source of epidural hematoma Foramen magnum Facial Bones discussed later Brain Anatomy Review : Brain Anatomy Review Occupies 80% of intracranial space Divisions Cerebrum Cerebellum Brain Stem Brain Anatomy Review : Brain Anatomy Review Cerebrum Cortex Voluntary skeletal movement level of awareness Frontal lobe Personality Parietal lobe somatic sensory input memory emotions Brain Anatomy Review : Brain Anatomy Review Cerebrum Temporal lobe speech center long term memory taste smell Occipital lobe origin of optic nerve Brain Anatomy Review : Brain Anatomy Review Cerebrum Hypothalamus center for vomiting, regulation of body temp and water sleep-cycle control appetite Thalamus emotions and alerting or arousal mechanisms Cerebellum coordination of voluntary muscle movement equilibrium and posture Brain Anatomy Review : Brain Anatomy Review Brain Stem connects hemispheres, cerebellum and SC responsible for vegetative functions & VS midbrain relay point for visual and auditory impulses pons conduction pathway between brain and other regions of body medulla oblongata cardiac, respiratory, and vasomotor control centers control of vomiting and coughing Brain Anatomy Review : Brain Anatomy Review Brain Stem Cranial Nerves Reticular Activating System level of arousal (level of consciousness) Primary control along with cerebral cortex Meninges dura mater: tough outer layer, separates cerebellum from cerebral structures, landmark for lesions arachnoid: web-like, venous vessels that reabsorb CSF pia mater: directly attached to brain tissue Brain Anatomy Review : Brain Anatomy Review Brain Stem Cerebral Spinal Fluid (CSF) clear, colorless circulates through brain and spinal cord cushions and protects ventricles center of brain secrete CSF by filtering blood forms blood-brain barrier Brain Metabolism & Perfusion : Brain Metabolism & Perfusion High metabolic rate consumes 20% of body’s oxygen largest user of glucose requires thiamine can not store nutrients Blood Supply vertebral arteries supply posterior brain (cerebellum and brain stem) carotid arteries most of cerebrum Brain Metabolism & Perfusion : Brain Metabolism & Perfusion Perfusion Cerebral Blood Flow (CBF) dependent upon CPP flow requires pressure gradient Cerebral Perfusion Pressure (CPP) pressure moving the blood through the cranium autoregulation allows BP change to maintain CPP CPP = Mean Arterial Pressure (MAP) - Intracranial Pressure (ICP) Brain Metabolism & Perfusion : Brain Metabolism & Perfusion Perfusion Mean Arterial Pressure (MAP) largely dependent on cerebral vascular resistance (CVR) since diastolic is main component blood volume and myocardial contractility MAP = Diastolic + 1/3 Pulse Pressure usually require MAP of at least 60 mm Hg to perfuse brain Intracranial Pressure (ICP) edema, hemorrhage ICP usually 10-15 mm Hg Mechanisms of Injury : Mechanisms of Injury Motor Vehicle Crashes most common cause of head trauma most common cause of subdural hematoma Sports Injuries Falls common in elderly and in presence of alcohol associated with subdural hematomas Penetrating Trauma missiles more common than sharp projectiles Categories of Injury : Categories of Injury Coup injury directly posterior to point of impact more common when front of head struck Contrecoup injury directly opposite the point of impact more common when back of head struck Diffuse Axonal Injury (DAI) shearing, tearing or stretching of nerve fibers more common with vehicle occupant and pedestrian Focal Injury limited and identifiable site of injury Head Injury : Head Injury Broad and Inclusive Term Traumatic insult to the head that may result in injury to soft tissue, bony structures, and/or brain injury Blunt Trauma more common dura intact fractures, focal brain injury, DAI Penetrating Trauma less common (GSW most common) dura and cranial contents penetrated fractures, focal brain injury Brain Injury : Brain Injury “a traumatic insult to the brain capable of producing physical, intellectual, emotional, social and vocational changes” Three broad categories Focal injury cerebral contusion intracranial hemorrhage epidural hemorrhage Subarachnoid hemorrhage Diffuse Axonal Injury concussion (mild and classic form) Causes of Brain Injury : Causes of Brain Injury Direct (Primary) Causes Impact Mechanical disruption of cells Vascular permeability or disruption Indirect (Secondary or Tertiary) Causes Secondary edema, hemorrhage, infection, inadequate perfusion, tissue hypoxia, pressure Tertiary apnea, hypotension, pulmonary resistance, ECG changes Pathophysiology of Brain Injury : Pathophysiology of Brain Injury As ICP and approaches MAP, cerebral blood flow Results in CPP Compensatory mechanisms attempt to MAP As CPP , cerebral vasodilation occurs to blood volume This leads to further ICP, CPP and so on Pathophysiology of Brain Injury : Pathophysiology of Brain Injury Hypercarbia causes cerebral vasodilation Results in blood volume ICP CPP Compensatory mechanisms attempt to MAP As CPP , cerebral vasodilation occurs to blood volume And, the cycle continues Hypotension results in CPP cerebral vasodilation Results in blood volume ICP CPP And, the cycle continues Pathophysiology of Brain Injury : Pathophysiology of Brain Injury Pressure exerted downward on Brain cerebral cortex or RAS altered level of consciousness hypothalamus vomiting brain stem BP and bradycardia 2° vagal stimulation irregular respirations or tachypnea unequal/unreactive pupils 2° oculomotor nerve paralysis posturing seizures dependent on location of injury Herniation Pathophysiology of Brain Injury : Pathophysiology of Brain Injury Levels of Increasing ICP Cerebral cortex and upper brain stem BP rising and pulse rate slowing Pupils reactive Cheyne-Stokes respirations Initially try to localize and remove painful stimuli Middle brain stem Wide pulse pressure and bradycardia Pupils nonreactive or sluggish Central neurogenic hyperventilation Extension Pathophysiology of Brain Injury : Pathophysiology of Brain Injury Levels of Increasing ICP Lower Brain Stem / Medulla Pupil blown (side of injury) Ataxic or absent respirations Flaccid Irregular or changing pulse rate Decreased BP Usually not survivable Pathophysiology of Brain Injury : Pathophysiology of Brain Injury Herniation transtentorial herniation downward displacement of the brain uncal herniation “downward displacement through the tentorial notch by a supratentorial mass exerting pressure on underlying structures including the brain stem” Head Injuries : Head Injuries Scalp Laceration/Avulsion Most common injury Vascularity = diffuse bleeding Generally does not cause hypovolemia in adults Can produce hypovolemia in children Head Injuries : Head Injuries Depressed Linear Stellate Basilar Skull Fractures Head Injuries : Head Injuries Linear Fracture Usually NOT identified in field 80% of all skull fractures Suspect based on Mechanism of injury Overlying soft tissue trauma Usually NOT emergency Temporal region = ~Epidural hematoma Head Injuries : Head Injuries Depressed Skull Fracture Segment pushed inward Pressure on brain causes brain injury Neurologic signs and symptoms evident Head Injuries : Head Injuries Basilar Skull Fracture Difficult to detect on x-ray Signs & Symptoms depend on amount of damage Diagnosis made clinically by finding: CSF Otorrhea CSF Rhinorrhea Periorbital ecchymosis Battle’s sign Head Injuries : Head Injuries Cerebrospinal Fluid Blood clotting delayed Halo sign Does not crust on drying Positive to Dextrostick Head Injuries : Head Injuries Basilar Skull Fracture Do NOT pack ears Let drain Do NOT suction fluid Do NOT instrument nose Head Injuries : Head Injuries Open Skull Fracture Cranial contents exposed Manage like evisceration Protect exposed tissue with moist, clean dressing (if possible) Neurologic signs & Symptoms evident Brain Injuries : Brain Injuries Intracranial Hematomas Epidural Subdural Intracerebral Brain Injuries : Brain Injuries Epidural Hematoma Blood between skull and dura Usually arterial tear middle meningeal artery Causes increase in intracranial pressure Brain Injuries : Brain Injuries Epidural Hematoma Unconsciousness followed by lucid interval Rapid deterioration Decreased LOC, headache, nausea, vomiting Hemiparesis, hemiplegia Unequal pupils (dilated on side of clot) Increase BP, decreased pulse (Cushing’s reflex) Brain Injuries : Brain Injuries Subdural Hematoma Between dura mater and arachnoid More common Usually venous bridging veins between cortex and dura Causes increased intracranial pressure Brain Injuries : Brain Injuries Subdural Hematoma Slower onset Increased ICP Headache, decreased LOC, unequal pupils Increased BP, decreased pulse Hemiparesis, hemiplegia Brain Injuries : Brain Injuries Intracerebral Hematoma Usually due to laceration of brain Bleeding into cerebral substance Associated with other injuries DAI Neuro deficits depend on region involved and size repetitive w/frontal lobe Increased ICP Brain Injuries : Brain Injuries Injury to Cerebral Parenchyma Laceration Concussion Contusion Brain Injuries : Brain Injuries Laceration Penetrating wounds GSW Stab Depressed Fracture Severe blunt trauma Sudden acceleration/deceleration Brain Injuries : Brain Injuries Concussion Transient loss of consciousness Retrograde amnesia, confusion Resolves spontaneously without deficit Usually due to blunt head trauma Head Trauma : Head Trauma Concussion Post-concussion syndrome Headaches Depression Personality changes Head Trauma Assessment : Head Trauma Assessment The Brain Is Enclosed In A Box Head Trauma Assessment : Head Trauma Assessment Early Detection/Control of Increased ICP Critical Head Trauma Assessment : Head Trauma Assessment Cerebral Perfusion Pressure = Mean Arterial Pressure - Intracranial Pressure CPP = MAP - ICP Head Trauma Assessment : Head Trauma Assessment LOC = Best Indicator Altered LOC = Intracranial trauma UPO Trauma patient unable to follow commands = 25% chance of intracranial injury needing surgery Head Trauma Assessment : Head Trauma Assessment Describe LOC changes based on response to environment Head Trauma Assessment : Head Trauma Assessment AVPU Scale A = Alert V = Responds to Verbal stimuli P = Responds to Painful stimuli U = Unresponsive Head Trauma Assessment : Head Trauma Assessment Glasgow Scale Eye Opening Motor Response Verbal Response Head Trauma Assessment : Head Trauma Assessment Glasgow Scale--Eye Opening 4 = Spontaneous 3 = To voice 2 = To pain 1 = Absent Head Trauma Assessment : Head Trauma Assessment Glasgow Scale--Verbal 5 = Oriented 4 = Confused 3 = Inappropriate words 2 = Moaning, Incomprehensible 1 = No response Head Trauma Assessment : Head Trauma Assessment Glasgow Scale--Motor 6 = Obeys commands 5 = Localizes pain 4 = Withdraws from pain 3 = Decorticate (Flexion) 2 = Decerebrate (Extension) 1 = Flaccid Head Trauma Assessment : Head Trauma Assessment Eyes Window to CNS Pupil size, equality, and response to light Head Trauma Assessment : Head Trauma Assessment Eyes Unequal Pupils + Decreased LOC = Compression of oculomotor nerve Probable mass lesion Unequal Pupils + Alert patient = Direct blow to eye, or Oculomotor nerve injury, or Normal inequality Head Trauma Assessment : Head Trauma Assessment Respiratory Patterns Cheyne Stokes Diffuse injury to cerebral hemispheres Central neurological hyperventilation Injury to mid-brain Apneustic Injury to pons Head Trauma Assessment : Head Trauma Assessment Respiratory Patterns Biot (Cluster) Injury to upper medulla Ataxic Injury to lower medulla Head Trauma Assessment : Head Trauma Assessment Motor Response Is patient able to move all extremities? How do they move? Decorticate Decerebrate Hemiparesis or Hemiplegia Paraplegia or Quadraplegia Head Trauma Assessment : Head Trauma Assessment Motor Response Lateralized/Focal Signs = Lateralized or Focal Deficits Altered motor function may be due to fracture/dislocation Head Trauma Assessment : Head Trauma Assessment Vital Signs Cushing’s Triad Suggests Increased Intracranial Pressure Increased BP Decreased Pulse Irregular respiratory pattern Head Trauma Assessment : Head Trauma Assessment Vital Signs Isolated head injury will NOT cause hypotension in adult Look for another life threatening injury Chest Abdomen Pelvis Multiple long bone fractures Head Trauma Assessment : Head Trauma Assessment Summary Most important sign = LOC Direction of changes more important than single observations Importance lies in continued reassessment compared with initial exam UPO, altered LOC in trauma = Intracranial injury Head Trauma Management : Head Trauma Management Airway Open Assume C-spine Trauma Jaw Thrust with C-spine Control Clear - Suction As Needed Maintain Intubation if No Gag Reflex, or RSI Avoid nasal intubation Head Trauma Management : Head Trauma Management Breathing Oxygenate - 100% O2 Ventilate No ROUTINE Hyperventilation Hyperventilate at 20 to 24 breaths per minute IF: Glasgow less than 8 Rapid neurologic deterioration Evidence of herniation Head Trauma Management : Head Trauma Management Hyperventilation--Benefits Decreased PaCO2 Vasoconstriction Decreased ICP Head Trauma Management : Head Trauma Management Hyperventilation--Risks Decreased cerebral blood flow Decreased oxygen delivery to tissues Increased edema Head Trauma Management : Head Trauma Management Circulation Maintain adequate BP and Perfusion IV of LR/NS TKO if BP normal or elevated If BP decreased LR/NS bolus titrated to BP ~ 90 mm Hg Consider PASG/MAST if BP below 80 Monitor EKG -- Do NOT treat bradycardia Head Trauma Management : Head Trauma Management Spinal motion restriction If BP normal or elevated, spine board head elevated 300 Head Trauma Management : Head Trauma Management Monitor for hyperthermia Vasoconstriction Heat retention Increased cerebral 02 demand Head Trauma Management : Head Trauma Management Drug Therapy Considerations Only after: Management of ABC’s Controlled hyperventilation Head Trauma Management : Head Trauma Management Drug Therapy Considerations Dexamethasone (Decadron®) Steroid Decreases cerebral edema Effects delayed Little usage today Head Trauma Management : Head Trauma Management Drug Therapy Considerations Mannitol (Osmitrol®) Osmotic diuretic Decreases cerebral edema May cause hypovolemia May worsen intracranial hemorrhage Often reserved for herniation Head Trauma Management : Head Trauma Management Drug Therapy Considerations Furosemide (Lasix®) Loop diuretic Decreases cerebral edema May cause hypovolemia Often reserved for herniation Head Trauma Management : Head Trauma Management Drug Therapy Considerations Diazepam (Valium®) Anticonvulsant Give if patient experiences seizures May mask changes in LOC May depress respirations May worsen hypotension Head Trauma Management : Head Trauma Management Drug Therapy Considerations Glucose Assess blood glucose Administer only if hypoglycemic Consider thiamine in malnourished Head Trauma Management : Head Trauma Management Transport Considerations Trauma Center GCS < 12 Evidence of herniation Unconscious Multisystem trauma with head trauma Consider comorbid factors Head Trauma Management : Head Trauma Management Helmet Removal Immediate removal if interferes with priorities access to airway or airway management ventilation cervical spine motion restriction May only need to remove face piece to access airway Consider interference with SMR Technique requires adequate assistance training in the procedure padding if shoulder pads left on You do not have the permission to view this presentation. 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